乙型流感IFA IgG熒光試劑盒

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乙型流感IFA IgG熒光試劑盒

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【公司名稱(chēng)】 廣州健侖生物科技有限公司
【市場(chǎng)部】    楊永漢

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【騰訊  】 2042552662
【公司地址】 廣州清華科技園創(chuàng)新基地番禺石樓鎮(zhèn)創(chuàng)啟路63號(hào)二期2幢101-103室

除了這項(xiàng)研究之外，去年來(lái)自加州大學(xué)舊金山分校的研究人員提出了一個(gè)相似的名稱(chēng)：CRISPRi，他們發(fā)現(xiàn)當(dāng)缺失核酸內(nèi)切酶活性的Cas9與一種導(dǎo)向RNA共表達(dá)時(shí)候，會(huì)產(chǎn)生一種DNA識(shí)別復(fù)合物，這種復(fù)合物能特異性干擾轉(zhuǎn)錄延伸，RNA聚合酶結(jié)合，或轉(zhuǎn)錄因子結(jié)合。
由此研究人員研發(fā)出了這種CRISPRi系統(tǒng)，這一系統(tǒng)能有效抑制大腸桿菌中靶向基因的表達(dá)，并且不會(huì)出現(xiàn)脫靶效應(yīng)。而且利用CRISPRi，還可以同時(shí)抑制多個(gè)靶基因，這種作用也是可逆。研究人員還證明，該系統(tǒng)也適用于哺乳動(dòng)物細(xì)胞中的基因表達(dá)抑制。
加州大學(xué)圣迭戈分校(University of California, San Diego)醫(yī)學(xué)院研究人員針對(duì)自閉癥研究出一套比較新銳的理論，并對(duì)其進(jìn)行了實(shí)際測(cè)試。研究結(jié)果顯示，自閉癥實(shí)際上是由異常的細(xì)胞通訊導(dǎo)致的，使用問(wèn)世已逾百年的治療嗜睡癥的藥物，可以恢復(fù)患有自閉癥的老鼠體內(nèi)的細(xì)胞活動(dòng)，消除老鼠的神經(jīng)失調(diào)癥狀。參與實(shí)驗(yàn)的老鼠年齡相當(dāng)于人類(lèi)30歲左右。相關(guān)研究成果在2014年6月17日的在線刊物《平移精神病學(xué)(Translational Psychiatry)》中發(fā)表。
醫(yī)學(xué)、兒科與病理學(xué)教授、羅伯特·納維奧(Robert K. Naviaux)博士為該研究的主要負(fù)責(zé)人，他表示：“這一發(fā)現(xiàn)與此前認(rèn)為的自閉癥是由內(nèi)部原因?qū)е碌睦碚撌制鹾?。在已知的與自閉癥有關(guān)的致病原因中，有20%的遺傳因素，但大部分病因并非如此。將基因與環(huán)境分離開(kāi)思考并不合理。實(shí)際上，基因與環(huán)境會(huì)互相作用，作用的結(jié)果就是新陳代謝。”
納維奧還提到：“自閉癥的普遍癥狀是代謝紊亂。細(xì)胞有代謝物環(huán)(halo of metabolites)和核苷酸環(huán)繞，所謂的代謝物環(huán)是由參與新陳代謝的微小分子組成的。”

In addition to the study, researchers from the University of California, San Francisco, last year proposed a similar name, CRISPRi, who found that a DNA lacking endonuclease co-expressed with a targeting RNA produced a DNA Recognition complexes that specifically interfere with transcriptional elongation, RNA polymerase binding, or transcription factor binding.
As a result, the researchers developed the CRISPRi system, which effectively inhibits the expression of the targeted genes in E. coli and does not show off-target effects. And using CRISPRi, you can also inhibit multiple target genes, this effect is also reversible. The researchers also demonstrated that the system is also suitable for gene expression inhibition in mammalian cells.
Researchers at the University of California, San Diego Medical School developed a set of cutting-edge theories of autism and conducted practical tests. The results show that autism is actually caused by abnormal cell communication, the use of more than a century has come out of the treatment of narcolepsy drugs, can restore autism in mice with cell activity in vivo, to eliminate symptoms of neurological disorders in mice . The age of the mice involved in the experiment is about 30 years old. Relevant research results are published in the June 17, 2014 online translation "Translational Psychiatry."
Robert K. Naviaux, MD, Ph.D., professor of medicine, pediatrics and pathology, is the lead investigator for the study. "The finding and the previously thought autism is due to internal causes Fit in. Among the known causes of autism-related causes of disease, there are 20% of genetic factors, but most of the causes are not the same genetically isolated from the environment is not reasonable thinking.In fact, the genes and the environment will be mutually Effect, the result of the role of metabolism. "
Navio also mentioned: "The most common symptom of autism is metabolic disorder, where cells have halo of metabolites and nucleotides that are made up of tiny molecules that are involved in metabolism."